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CV and CVA

Colleen Nikstenas

Review questions and answers for our Cardiovascular unit with venous, arterial, cerebrovascular and hypertension.

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Across
5.ischemic or hemorrhagic
9.quivering heartbeat or dysrhythmia involving upper chambers
10.Treatment given for preventative purpose
11.volume of blood ejected from heart with each beat
12.main goal of CVA interventions aside from patient safety: increase this safely and preserve it
15.Tougher, denser, cleaner cholesterol
18.Furosemide
19.buildup of plaques from lipoproteins within artery walls at sites of inflammation or injury; causes hardening
20.amount of blood ejected from one ventricle per heartbeat
22.most common cause is rheumatic fever or other bacterial infection
23.Narrowing of vessel or valve
24.hallmark signs: crackles, pink frothy sputum
25.may have concurrently or comorbid with CHF; possibly causing or as a result of
26.CHF medication and antiarrhythmic to decrease rate
31.Sign of right sided heart failure, fluid volume overload
32.aneurysm shape that bulges on all sides of vessel
34.Antidote for heparin
37.traceable to other conditions
40.common cause of this condition is intravenous drug use
41.<150 mg/dL
43.Valve repair
45.issue that can cause inflammation throughout the body; risk factor for all CV issues
47.Intervention for fluid volume overload, heart failure
48.Potassium sparing diuretic
49.Lols
50.Dill pickles in milk with "ball peen hammers" (pines and dils)
53.Disorder characterized by impaired circulation to fingers and toes; patient should not wear constrictive clothes or smoke
54."ballooning" of heart with thinned and weakened walls
55.Heart failure side with systemic effects
56.severe infection that can lead to endocarditis, valve disease; caused by strep infections
58.common condition of enlargement with diminished room for blood to fill ventricles
Down
1.Renin-angiotensin-aldosterone system
2.Side effects include worst headache from a medication, drop in BP, and decreased oxygen need of heart; used for angina
3.Thickened and stiffened arterial walls; often comorbid with or caused by atherosclerotic changes
4.Heparin given to prevent clots, usually via IV route
5.the product of stroke volume and peripheral resistance
6.Chest pain or discomfort connected with cardiovascular occlusions or spasms
7.BP over 140/90, patient under 65
8.Diet, lifestyle, activity, medications
13.occlusion or blockage of bloodflow leads to tissue damage
14.no known cause
16.Left sided heart failure effects
17.Thiazide diuretic
18.Enoxaparin and Dalteparin
21.protein released by brain to increase diuresis with increased sodium levels
27.Stiffened and fibrotic scarring leads to difficulty filling, shape changes
28.primary assessment of fluid balance in CHF; taken same time daily with same equipment; no increase is good.
29.Age, race, genetics, gender
30.MVR
33.Ring surrounding valve
35.VTE mechanical prophylaxis
36.resistance left ventricle has to overcome to circulate (peripheral resistance)
38.blood remains in artery, possibly between injured layers causing bulge
39.inflammation of sac that contains heart
42.volume of blood in ventricles at end of diastole
44.Ballooning of leaflet into chamber beyond or behind causing regurgitation of flow
46.reddish-blue discoloration of extremities; related to PAD
51.Antidote for warfarin
52.Sticky, spongy, fatty cholesterol
57.congestive heart failure

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