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Across 3. Vaughan Williams class IV is a ____ blocker. 7. ________, an unclassified antidysrhythmic has a half life of less than 10 seconds. 8. Vaughan Williams class II is a _____ blocker. 11. Lidocaine has extensive ______ metabolism and doses are reduced by 50% for liver failure or ETOH patients. 12. _____ juice has a negative interaction with quinidine and increases risk of cinchonism. 14. Lidocaine mixed with epinephrine should not be given _________. 16. Quinidine causes ________, which is tinnitus, loss of hearing, or slight blurring of vision, 18. To decrease _ _ upset, oral doses can be taken with food and fluids. 19. _____ release should not be crushed or chewed. 21. N/V/D, ______, headache, & blurred vision are the adverse effects of antidysrhythmics 22. _______ means antidysrhythmics can make an existing dysrhythmia worse. 23. Significant side effects of this 1b drug are: cardiac asystole, ventricular ectopic beats. 26. Vaughan Williams class III drug prolongs_____ ______.

Down 1. Never _____ up the dose or omit a dose. 2. ____ intake should be limited or avoided. 4. Deviation from normal rhythm 5. Adverse effects of ______, 1a drug, are N/V/D and should not be used with patients who have lupus. 6. After administering an antidysrhythmic, provider should be notified if syncope, dizziness, blurred vision, ____, or change in respiratory status. 9. No rhythm 10. _____, class 1b drug and, is the most effective for treatment of ventricular dysrhythmias. 13. With adiodarone, ________ is an adverse effect. 15. Decreased cardiac output and risk for injury are ND for _______ drugs. 17. Postural _____ is a common adverse effect, so patients should change positions slowly. 20. Vaughan Williams class I is a ______ blocker. 24. Consumption of a high-_____ diet is recommended to minimize GI adverse effect of antidysrhythmic 25. After the drug is absorbed a ____ matrix may pass through the stool.